By D. T. Kanazir (auth.), Oddvar F. Nygaard, Arthur C. Upton (eds.)

ISBN-10: 1461367182

ISBN-13: 9781461367185

ISBN-10: 1461538505

ISBN-13: 9781461538509

The dramatic reduce within the occurrence of abdominal melanoma in industrialized international locations in the past 50 years, that is but to be absolutely defined, and the commentary that carcinogenesis in laboratory animals may be inhibited by way of antioxidants, retinoids, and caloric limit, between different affects, problem us to press on within the look for useful potential to avoid melanoma. it really is with regards to this objective that the stories summarized during this e-book have particular value. This ebook relies at the invited and contributed papers offered on the 3rd Internationai convention on Anticarcinogenesis and Radiation security, hung on October 15-21, 1989, in Dubrovnik, Yugoslavia. They hide a large diversity of investigations into the mechanisms and inhibition of carcinogenesis. based on fresh advances in our knowing of the significance of oncogenes and tumor-suppressor genes in carcinogenesis, a number of the stories specialise in mutations and similar alterations on the point of DNA. whilst, notwithstanding, different stories care for dietary, immunological, endocrinological, and epidemiological features. In all, a number of the experiences tackle carcinogenesis and its inhibition at nearly each point of organic association. incorporated during this compendium are well timed stories of various and promising examine techniques for melanoma prevention, as pursued via investigators in numerous elements of the realm. The Organizers of the convention are thankful to the numerous scientists who've contributed to the amount, in addition to to the sponsors of the convention, with no whose beneficiant help this booklet don't have been possible.

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It develops selective toxicity against these cells (16), based on an inhibition of mitochondrial bioenergetic function (17). This effect is enhanced in vivo in tumor-bearing mice by 2-deoxyg1ucose, an inhibitor of glycolysis (18,19). These inhibitors of bioenergetic functions are aimed at the killing of cancer cells by energy depletion rather than by destruction of vital gene functions. Therefore, these and similar drugs should have a reduced risk for the induction of treatment-induced secondary cancer.

F. C. Upton. Plenwn Press. New York. 1991 33 Table 1. The Frequency of Secondary Tumors in Patients with Primary Tumor of the Most Frequent Localizations in our Material Localization of Primary Tumors Breast PVU Lung Larynx No. of Patients wi th Primary Tumors No. 66% tumor caused or at least facilitated the appearance of the new tumor, but also whether the conditions for the appearance of the first tumor persisted, and also favored the appearance of the second tumor. During the period January 1st, 1981 to December 31st, 1987, we examined patients with the diagnosis of malignant disease.

Effects of time interval between first and second primary tumors. Br. J. Cancer 58:191194 (1988). J. Bailey, C. P. T. J. -C. Gazet, Bilateral carcinoma of the breast. ~ ~ Surg. ~:514-516 (1980). T. Ricsz, Z. Risko, V. Winkler and J. Juhasz, A clinico-pathological study on the correlation of immunosupression and mUltiple primary malignant neoplasmas. Neoplasma ~:409-420 (1976). G. Moertel, MUltiple primary malignant neoplasms: Their incidence and significance. Recent Results Cancer Res. 7:1-108 (1966).

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Anticarcinogenesis and Radiation Protection 2 by D. T. Kanazir (auth.), Oddvar F. Nygaard, Arthur C. Upton (eds.)

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